Our latest scientific tests point out that this is due to faulty trafficking of late endosomes to lysosomes, with concomitant homotypic fusion in the affected vesicular compartments [five]. The defect in lysosome-directed trafficking also affects autophagic flux, with resultant accumulation of autophagosomes [5]. Ultimately, the integrity from the mobile membrane https://pauld196vae9.blogpayz.com/profile